Changes
/* Cardiovascular diseases */
Inflammation is one of the key processes linked to various diseases and disorders. There are many opportunities for designing and developing specific new drugs for inflammatory responses.
[[image:market.png|center|350 1200 px|thumb|[http://www.chromos.com/documents/roadshowsept14_003.pdf Source]]]
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Inflammation is characterized by:
* increased blood flow to the tissue causing
* increased temperature,
* redness,
* Several angiogenic and myogenic stem cell therapies are under investigation and promise in the long run to advance cardiac rehabilitation and even prevent heart failure. Despite the development of improved therapies, millions of Americans continue to live with life-threatening cardiovascular diseases.
[[image:cardiovascilar diseases.png|center|thumb|800 px2000px| [http://www-ermm.cbcu.cam.ac.uk/0500918Xh.htm Source]]]
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==Drug targets==
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<br>
===C-reactive protein (CRP)===
Interactive signaling pathway of G- protein represented below with information on few patents targeting different sites. Click on the Hypertext molecules in the main pathway, to visualize the patent details.
<center><gflash>800 800 http://www.dolcera.com/website/assets/G-protein.swf</gflash></center>
==Research activities==
Recent research progress (year 2007) in the area of G-proteins is illustrated below, with interactive gene linking.
<center><gflash>800 800 http://www.dolcera.com/website/assets/Gene-Interaction.swf</gflash></center>
===Future potential areas===
# In vivo activation of '''G{alpha}q/PKC pathways'' could upregulate GRK2 expression in cardiovascular cells, thus contributing to the increased desensitization of ß-adrenergic and other GPCR systems, is of potential physiological relevance. Detailed mechanisms of regulation of GRK2 expression in different types of cells of the cardiovascular system, particularly in cardiac cell lines, and the determination of possible changes in GRK2 levels in VSMCs of patients with cardiovascular diseases are interesting fields for future research and may help to develop new therapeutic strategies based on the modulation of GRK2 expression. [http://circ.ahajournals.org/cgi/content/full/circulationaha;101/17/2083]]
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